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doi: 10.1007/s40257-022-00677-0 79

Recent evidence suggests there is also likely an HDL-independent pathway for systemic cholesterol removal through transintestinal cholesterol excretion (TICE) (711)

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Critically, we identify that Np63 transcriptionally activates these enzymes, creating a metabolic dependency that enables tumor cells to evade carboplatin-induced cytotoxicity

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The comparison of field and test bench measurements given in Fig